In this video we discuss botox for hair loss – how it helps relieve scalp tension which is the root cause of hair loss, and show studies where patients regrew hair from botox treatment.
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You’ve likely heard about botox injections for aesthetic reasons, whether from friends and family, in regards to your favorite reality television stars, or on the news.
But even more than just reducing the appearance of fine lines and wrinkles, botox injections can also be an effective treatment for pattern hair loss.
This article will first introduce a more modern theory of hair loss – the scalp tension theory. It’ll then delve into the use of botox for hair growth, as well as the science behind this latest treatment option.
2 studies showed that botox had very similar hair regrowth results as taking finasteride, but without the side effects that are associated with pharmaceutical DHT blockers.
By the end of this article you’ll know just how effective botox is for treating male pattern baldness, and if it could be a good option for you.
And approximately how much it could cost.
Unfortunately, there currently aren’t any clinicians performing this procedure. However if you’re interested in finding out when the procedure becomes available in your nearest city, you can join the botox waiting list below.
An Introduction to Botox for Hair Growth
Botox, also known as Botulinum Toxin (BTX), is an injection prepared from the bacterial toxin botulin (1).
Botox injections are used medically in the treatment of muscular disorders, as well as cosmetically to remove fine lines and wrinkles in the face, neck, and other parts of the body.
In more recent years, botox injections have also been found to possibly treat an unlikely condition: Androgenetic Alopecia (AGA), also known as male-pattern baldness.
To understand how botox injections may induce hair growth, however, you’ll first need to understand the scalp tension theory of hair loss.
Scalp Tension: A New Theory on Pattern Hair Loss
It’s widely believed that the androgen hormone, DHT, is the sole cause of hair loss in men and women with androgenetic alopecia.
But there’s more and more evidence coming to light which suggests that DHT may not play such a large role. In fact, it may not even be a cause of hair loss but, instead, a side effect of the true cause.
So, what’s that “true” cause? Scalp tension.
Let me explain.
According to this theory, the galea muscle (which is connected to the top-layer of skin by a “fibrous rigid subcutaneous layer”) plays a significant role in the progression of pattern balding. This is because the galea muscle is often affected by physical stressors, such as skull bone growth or tensing of the scalp, face, and neck muscles (2, 3).
Interestingly, this muscle rests right where pattern baldness tends to occur. That is, from behind the ears to the hairline and temples.
But what is it about scalp tension that seems to trigger hair loss?
According to researchers, the tension may induce a “pro-inflammatory cascade … which induces TGF-β1 alongside increased androgen activity (5-αR2, DHT, and AR), which furthers TGF-β1 expression in already-inflamed AGA-prone tissues (2).”
TGF-β1, or Transforming Growth Factor Beta 1, actually plays an important role in the hair growth process. But TGF-β has also been shown to increase the activity of an enzyme called alkaline phosphatase which contributes to scalp calcification (4).
With an increased presence of TGF-β and androgens, then, the hair follicles are unable to operate as they should (due to inflammation, lack of blood flow, and calcification) and this leads to hair thinning and loss.
The interesting thing about this model is that it doesn’t reject the idea of genetic predisposition.
It does, however, refute the belief that “AGA-prone follicles are genetically programmed to become sensitive to DHT.”
The scalp tension model also answers a number of questions which the DHT model seems to be unable to answer. These include:
Why DHT increases in AGA-prone scalp tissues (i.e., DHT is a response to tension-mediated inflammation)
The mechanisms by which DHT is involved in AGA progression (i.e., DHT is involved in the onset of fibrosis and calcification)
The pattern of AGA (i.e., AGA progression matches that of where GA-transmitted scalp tension is highest, and progresses as peak tension points change during fibrosis onset)
Why AGA is observed more often in elderly populations versus young adults (i.e., calcification and fibrosis have had more time to accumulate)